Some information and I only looked it up as I am currently working on my dissertation.
Now, legally I cannot post the articles as it comes as a subcription, however they are fully referenced, so you are more than welcome to go and find them, should the synopsis not be enough. I also add that that I am pro choice and one does the best for their baby, but I reiterate, giving a rusk is like feeding a little one a hob nob. There are much better foods to offer.
Sample:
40 Children
Aged Between 4-13 presenting with complaints of recurrent diarrhea, abdominal distention, severe emaciation and dehydration were included.
Conclusion:
Majority of children with Celiac disease presented with typical symptom, while Celiac crisis was characterized by severe dehydration, weakness and calcium deficiency signs. Most were the product of consanguineous marriages and were given Gluten-containing weaning foods as early as the 4th month of life.
Celiac crisis presented with profuse diarrhea, severe dehydration; abdominal distention; pedal edema, carpopedal spasm due to tetany; wasted muscles; head drop and inability to stand. The serum TtG antibodies in thirty-eight cases (95%) were above the cut off level of 7 u/ml ranging from 35-99 u/ml. The histopathology of specimens from distal duodenum revealed lesions of M3 type in thirteen (32.5%) and M2 type in eighteen cases (45%). All cases recovered and improved on follow-up after strict adherence to gluten-free diet (GFD).
Babar, M, Ahmad, I, Rao, M, Iqbal, R, Asghar, S, & Saleem, M 2011, 'Celiac disease and celiac crisis in children', Journal Of The College Of Physicians And Surgeons--Pakistan: JCPSP, 21, 8, pp. 487-490
DESIGN, SETTING, AND PATIENTS:
Prospective observational study conducted in Denver, Colo, from 1994-2004 of 1560 children at increased risk for celiac disease or type 1 diabetes, as defined by possession of either HLA-DR3 or DR4 alleles, or having a first-degree relative with type 1 diabetes. The mean follow-up was 4.8 years.
MAIN OUTCOME MEASURE:
Risk of CDA defined as being positive for tissue transglutaminase (tTG) autoantibody on 2 or more consecutive visits or being positive for tTG once and having a positive small bowel biopsy for celiac disease, by timing of introduction of gluten-containing foods into the diet.
RESULTS:
Fifty-one children developed CDA. Findings adjusted for HLA-DR3 status indicated that children exposed to foods containing wheat, barley, or rye (gluten-containing foods) in the first 3 months of life (3 [6%] CDA positive vs 40 [3%] CDA negative) had a 5-fold increased risk of CDA compared with children exposed to gluten-containing foods at 4 to 6 months (12 [23%] CDA positive vs 574 [38%] CDA negative) (hazard ratio
, 5.17; 95% confidence interval [CI], 1.44-18.57). Children not exposed to gluten until the seventh month or later (36 [71%] CDA positive vs 895 [59%] CDA negative) had a marginally increased risk of CDA compared with those exposed at 4 to 6 months (HR, 1.87; 95% CI, 0.97-3.60). After restricting our case group to only the 25 CDA-positive children who had biopsy-diagnosed celiac disease, initial exposure to wheat, barley, or rye in the first 3 months (3 [12%] CDA positive vs 40 [3%] CDA negative) or in the seventh month or later (19 [76%] CDA positive vs 912 [59%] CDA negative) significantly increased risk of CDA compared with exposure at 4 to 6 months (3 [12%] CDA positive vs 583 [38%] CDA negative) (HR, 22.97; 95% CI, 4.55-115.93; P = .001; and HR, 3.98; 95% CI, 1.18-13.46; P = .04, respectively).
CONCLUSION:
Timing of introduction of gluten into the infant diet is associated with the appearance of CDA in children at increased risk for the disease.
JAMA. 2005 May 18;293(19):2343-51.
Risk of celiac disease autoimmunity and timing of gluten introduction in the diet of infants at increased risk of disease.
I edit to say that there are better articles out there, however my subscription is not scientific, and more humanities based, so the access is limited. If anyone has any more?
